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I am a 33 year old male. I had been using Propecia for 3 years and switched to Dutasteride approximately 6 months ago. Since switching to Dutasteride, I am experiencing less sexual side effects than Propecia, even at 1.0mg daily dose (twice the dose which is recommended in treating BPH) – I am also experiencing more hair regrowth than Propecia. I am wondering how and where you draw the inference that Dutasteride has the potential to cause sterility. Any firm evidence, clinically?

Just like your positive hair growth experience with dutasteride there are others who have negative experience with sterility and sexual side effects.

I cannot find a study or firm evidence, but I always try to balance risks and benefits. If you are never planning to have children, then it may not matter but if you are, why would you want to risk it? In addition dutasteride is not approved for the treatment of genetic balding and there is also no clear evidence it is much better than Propecia (despite your personal experience). If you want to know if you now have a problem, your doctor can order a sperm count which will show a problem if there is one.

Does fungal infection if not treated mimic Male pattern baldness say (norwood 5)?. Could u explain types of infections or fungal infections in general a little better?? would we see hair loss in patches or one might start losing hair from the front due to infection to all the way back? if treated do these conditions reverse back to having a full head of hair??

Simply put, if you let the fungal infections go on for years, it might look like patterned hair loss to the inexperienced eye. For more on fungal infections, see Wikipedia – mycosis.

Dear Dr. Rassman: Had the 2nd of my three procedures today. Almost like a sculpture, it really now has started to take shape. Sure, I’ve looked at photos and seen real live guys at your Open House shows who’ve had this process, but there’s nothing like having SMP done to your own challenged head.

Once again, Kudos to the technicians who worker harder than stainless steal and have the patients of angels and Dr. Pak who stepped in mid-way and said: “Now it’s time to mail down your front hairline” which he did with the precision of a rocket shop landing on the moon.

But I do have one regret… which I expressed to your staff openly: “Where were you guys 40 years ago when I first coulda used this?

Dr. Rassman’s comment: This man started his hair transplant process too many years ago. He is now in his 60s and shaved his head for the procedure. He will eventually allow his hair to grow on top of the SMP. This type of comment occurs almost every day in our office. Scalp MicroPigmentation is one of the most satisfying procedures we have ever offered.

I read an article a while back that said resaerchers had found that acne was actually caused by a bacteria or fungal infection within the pores not an internal cause as previously thought.

Could it be, with Hair loss that resaerchers are only focusing on internal causes such as the DHT and actually overlooking other external causes like some kind of Fungal Folliculitis of the scalp that eats away at the folicles? Where does the DHT come from and why can’s we stop it?

Anything is possible, but the explanation you offered has not come to the forefront. I would not be surprised if there were other mediators for hair loss that make hair loss show up in one person and not another. We are not well informed enough to go there just yet.

This is what we know (taken from an article by Dr. David Whiting, a widely acknowledged leading researcher/dermatologist): “Testosterone and dihydrotestosterone can circulate systemically to follicles, or be manufactured locally in the follicle from circulating weak androgens (dehydroepiandro-sterone and androstenediol) via complex enzyme-mediated processes involving specific dehydrogenase and reductase enzyme pathways. All of these enzyme reactions are dependent upon specific pyridine cofactors. It is clear that reductase, dehydrogenase, and probably aromatase enzymes are of major importance in hair growth as they mediate the complex interchange of sex hormones implicated in anagen activity.”

That is the biochemistry of DHT (dihydrotestosterone).

This is an inherited form of hair loss that is often on both sides impacting the temple peak area and is easily fixed with hair transplant (the only solution)

The hair is a rapidly growing organ, possibly among the fastest of body well (outside the intestine). Cancers are also fast growing so when the cancer is treated with Chemotherapy, the treatment targets fast growing cells. That is why many people who get this get stomach and intestinal side effects. Hair loss occurs in the same way. We have written about the Cold Cap to prevent hair loss with chemotherapy and it is an important tool, review posts here: https://baldingblog.com/?s=cold+cap&x=0&y=0 and you can review the many posts on the subject.

Some people only partially regrow their hair if they lost it. This may be caused by permanent changes in the life of the hair follicle which is frequently limited anyway. When a hair follicle reaches the end of its life (from genetic balding or even old age), the cells undergo apoptosis (cell death). The Chemotherapy may damage the hair cycle clock limiting how many cycles the hair goes through. We know that hair goes through aging in all of us and we often see changes of hair character (from a coarser hair to a finer hair) or graying (as we lose the pigment cells that give our hair color.